Gabtin is an anti-seizure (anti-convulsant) drug that is used for preventing seizures and for treating post-herpetic neuralgia, the pain that follows an episode of shingles. Doctors do not know how Gabtin works (the mechanism of action). Gabtin structurally resembles the neurotransmitter gamma aminobutyric acid (GABA). (Neurotransmitters are drugs that nerves use to communicate with one another.) It is possible that this similarity is related to Gabtin's mechanism of action. In animal models used for testing the anticonvulsant and analgesic (anti-pain) activities of drugs, Gabtin prevents seizures and reduces pain-related responses.
Other names for this medication:
Gabapentin,
Neurontin,
Algia,
Alpentin,
Bapex,
Blugat,
Dineurin,
Edion,
Epleptin,
Gabagamma,
Gabalept,
Gabanet,
Gabaneural,
Gabantin,
Gabapen,
Gabapentina,
Gabapentine,
Gabapin,
Gabaran,
Gabatal,
Gabateva,
Gabatin,
Gabatine,
Gabator,
Gabax,
Gabex,
Gabexal,
Gabictal,
Gabiton,
Gaboton,
Gabrion,
Gabtin,
Galepsi,
Ganin,
Gantin,
Gapridol,
Gatilox,
Gordius,
Kaptin,
Neurexal,
Neuril,
Neuropen,
Neuros,
Neurotin,
Nupentin,
Pendine,
Progresse,
Ritmenal,
Tebantin,
Similar Products:
Mestinon,
Efamat,
Cefodox,
Gabex,
Ticamet,
Biclar,
Bioprim,
Micoral,
Lopram,
Noroxine,
Tolbin,
Gastronorm,
Levotac,
Rantec,
Firide,
Tersen,
Duodart,
Dalsan,
Ditrim,
Lopraxer,
Nidagyl,
Proximax,
Vensir,
Thelmox,
Promat,
Mediklin,
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In this study, we identified a variable region in the C-terminus of hemocyanin from the shrimp Litopenaeus vannamei (2288-2503bp, HcSC) by sequence alignments. Dating violence in young people is highly prevalent, and bidirectional violence characterizes most violent relationships. Effects of ion transport inhibition on rat mandibular gland secretion. A series albenza manufacturer of Tenm4 mouse alleles showing a broad range of phenotypes were isolated after ENU mutagenesis.
We searched the Cochrane Neuromuscular Disease Group Specialized Register (15 October 2012), CENTRAL (2012, Issue 9 in The Cochrane Library), MEDLINE (January 1966 to October 2012), EMBASE (January 1980 to October 2012), AMED (January 1985 to October 2012), CINAHL Plus (January 1937 to October 2012) and LILACS (from January 1982 to October 2012). During fermentative melocin drug abuse growth, the lack of either PYC gene had little effect on the level and pattern of expression of the other PYC gene, indicating further their separate regulation.